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이런 연구들은 AGEs 와 progression of osteoarthritis 의 직접적인 원인규명은 아직 못함
이는 Willet et al 연구에서도 지적
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However, as pointed out by Willet et al, these studies have yet to show a direct causative effect between AGEs and theprogression of osteoarthritis.
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본연구에서 다루지 못한것-향후 연구에 떠넘길때
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However, this is beyond the scope of this study and should be considered for future work.
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Obesity 와 advanced accumulation of the AGEs 가 osteoarthritic phenotype 에 영향
하지만 어떻게 그런지는 알수없음
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Both obesity and the advanced accumulation of the AGEs may contribute to the osteoarthritic phenotype; however, the manner of this still remains unclear.
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본연구-ajoene는 LKB1을 통해서AMPK를 활성화함, CaMKK는 아님
이는 LKB1가 ajoene에 의해 유도된 AMPK 활성화의 upstream kinase 역할을 한다는 의미로 보여진다.
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In the present study, ajoene activated AMPK through LKB1, but not CaMKK, implying that LKB1 many serve as an upstream kinase of AMPK activation elicited by ajoene.
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